Knee joint laxity can result from soft tissue injury, such as an anterior cruciate ligament (ACL) tear, or genetic factors such as joint hypermobility syndrome (JHS). The degree of a subject’s knee laxity along a continuous spectrum depends on the mechanical properties of the structures, and increased motion that typically follows joint injury. At some threshold along the continuum, instability becomes pathologic and the risk of further joint injury increases.

This content is only available via PDF.
You do not currently have access to this content.